Dementia in idiopathic Parkinson's disease
Identifieur interne : 002979 ( Main/Exploration ); précédent : 002978; suivant : 002980Dementia in idiopathic Parkinson's disease
Auteurs : S. Elizan [États-Unis] ; Hava Sroka [Israël] ; H. Maker [États-Unis] ; H. Smith [États-Unis] ; D. Yahr [États-Unis]Source :
- Journal of Neural Transmission [ 0300-9564 ] ; 1986-09-01.
Abstract
Summary: A series of 203 patients with primary Parkinson's disease treated with L-DOPA, with adequate neurological documentation of mental status at serial intervals during their illness, constitutes the study population. Based on the results of the latest neurological examination, slightly less than one-third (29%) had mental impairment assessed as neurologically significant. Of the eleven clinical variables analysed (Cox regression analysis) for potential influence on the occurrence of an organic mental syndrome, four had a statistically significant effect: (1) the stage of disease at initial neurological examination; (2) the occurrence of acute confusional states; (3) the years of Parkinson's prior to L-DOPA; and (4) the total duration of L-DOPA therapy. The pathogenesis of dementia in this subgroup of Parkinson's disease is discussed.
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DOI: 10.1007/BF01249089
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Yahr</name></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:16D1984518DEBE6FC45DDB874AA69419C46427A9</idno><date when="1986" year="1986">1986</date><idno type="doi">10.1007/BF01249089</idno><idno type="url">https://api.istex.fr/document/16D1984518DEBE6FC45DDB874AA69419C46427A9/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">001A73</idno><idno type="wicri:Area/Main/Curation">001814</idno><idno type="wicri:Area/Main/Exploration">002979</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Dementia in idiopathic Parkinson's disease</title><author><name sortKey="Elizan, S" sort="Elizan, S" uniqKey="Elizan S" first="S." last="Elizan">S. Elizan</name><affiliation wicri:level="2"><country>États-Unis</country><placeName><region type="state">État de New York</region></placeName><wicri:cityArea>Departments of Neurology, The Mount Sinai Medical Center, New York</wicri:cityArea></affiliation></author><author><name sortKey="Sroka, Hava" sort="Sroka, Hava" uniqKey="Sroka H" first="Hava" last="Sroka">Hava Sroka</name><affiliation wicri:level="1"><country xml:lang="fr">Israël</country><wicri:regionArea>Department of Neurology, Bedinson Medical Center, Petah Tikva</wicri:regionArea><wicri:noRegion>Petah Tikva</wicri:noRegion></affiliation></author><author><name sortKey="Maker, H" sort="Maker, H" uniqKey="Maker H" first="H." last="Maker">H. 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Yahr</name><affiliation wicri:level="2"><country>États-Unis</country><placeName><region type="state">État de New York</region></placeName><wicri:cityArea>Departments of Neurology, The Mount Sinai Medical Center, New York</wicri:cityArea></affiliation></author></analytic><monogr></monogr><series><title level="j">Journal of Neural Transmission</title><title level="j" type="sub">Basic neurosciences and genetics, Parkinson's disease and allied conditions, Alzheimer's disease and related disorders, biological psychiatry</title><title level="j" type="abbrev">J. Neural Transmission</title><idno type="ISSN">0300-9564</idno><idno type="eISSN">1435-1463</idno><imprint><publisher>Springer-Verlag</publisher><pubPlace>Vienna</pubPlace><date type="published" when="1986-09-01">1986-09-01</date><biblScope unit="volume">65</biblScope><biblScope unit="issue">3-4</biblScope><biblScope unit="page" from="285">285</biblScope><biblScope unit="page" to="302">302</biblScope></imprint><idno type="ISSN">0300-9564</idno></series><idno type="istex">16D1984518DEBE6FC45DDB874AA69419C46427A9</idno><idno type="DOI">10.1007/BF01249089</idno><idno type="ArticleID">Art12</idno><idno type="ArticleID">BF01249089</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0300-9564</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Summary: A series of 203 patients with primary Parkinson's disease treated with L-DOPA, with adequate neurological documentation of mental status at serial intervals during their illness, constitutes the study population. Based on the results of the latest neurological examination, slightly less than one-third (29%) had mental impairment assessed as neurologically significant. Of the eleven clinical variables analysed (Cox regression analysis) for potential influence on the occurrence of an organic mental syndrome, four had a statistically significant effect: (1) the stage of disease at initial neurological examination; (2) the occurrence of acute confusional states; (3) the years of Parkinson's prior to L-DOPA; and (4) the total duration of L-DOPA therapy. The pathogenesis of dementia in this subgroup of Parkinson's disease is discussed.</div></front></TEI><affiliations><list><country><li>Israël</li><li>États-Unis</li></country><region><li>État de New York</li></region></list><tree><country name="États-Unis"><region name="État de New York"><name sortKey="Elizan, S" sort="Elizan, S" uniqKey="Elizan S" first="S." last="Elizan">S. 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